Diabetes is a term for glucose intolerance where the body cannot adequately manage the blood glucose levels. This is usually caused by insulin resistance (intolerance), reduced levels of insulin or the absence of insulin. Insulin is the hormone secreted by the pancreas that helps regulate blood glucose levels. When insulin is secreted, the body cells increase the uptake of glucose from the blood and excess glucose is converted into glycogen and stored in the liver and muscle cells. Any glucose above and beyond may be converted into fat and stored in fat cells.

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“Type-2 Diabetes” is primarily responsible for a combination of abnormalities. Type-2 diabetes is a typical body condition in which the body cells are less reactive to the insulin produced by the body. Because of the body is less reactive to the insulin, it is necessary for the body to produce more insulin to maintain the normal metabolism.

Type-2 diabetes normally is not the sole result of the insulin resistance itself. To balance the resistance the pancreas will produce more insulin. The blood sugar levels will be at normal level in most of the people with insulin resistance. In some people, the pancreas fails to compensate the increasing need of insulin. In such cases the blood sugar level increases and this deficiency in the supply of insulin causes ‘type-2 diabetes’.

Type 2 Diabetes can best be understood as a problem arisen because of a considerable gap that is created in body in terms of deficient supply against the demand of insulin. The pancreas cannot cope up with increasing demand of insulin with insulin resistance as it produces too little insulin. The puzzle to be solved is the contribution of overweight for insulin resistance. According to studies fat cells are not just storage sites, it also produce fatty acids and the secretion of proteins such as leptin, resistin and adiponectin which obstructs the secretion and action of insulin in the body.

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